balla
04-09-2006, 05:51 PM
PLEASE CAN ANYONE HELP AND GIVE ADVICE ON THESE MED RECORDS.
rofessor Mitchell?s findings,
22nd may 1998,
Thought to have had vaso-vagal when taken into Furness general in 1998.
Also optic discs swollen more on left than right.
11th June 1998
Oedema of both discs, left side is showing significant field defect and some reduction in colour vision, and VEP showed delayed conduction on both visual pathways.
B scan suggested optic nerve cyst on left side, and possible nerve head drusen on right
22nd June 1998
Thinking the eye problem could be optic nerve head drusen, and pupilay changes may be related to inflammatory uveitis
29th June 1998,
After 15 days in hospital, possible pseudo papilloedema, due to optic head drusen.
Significant field defects on left side, bilateral full discs with blurred margins.
No reaction to light as well as accommodation.
30th July 1998
Visual evoked potentials mildly delayed,
Oligoclonal bands found in CSF and these are absent from the serum.
Raising the possibility of M.S.
9th July 1998
Optic nerve drusen could be anterior segment inflammation.
10th August 1998,
Some indication of demylination. Could be high pressure around optic nerve.
Iris atrophy showing itself as transillumination. Ocular albinism but think its related to chronic inflammation.
Fluoroscein showed leakage from the discs.
11th September 1998.
Pupil abnormality is related to low-grade chronic inflammation.
Possible granulomatous disorder.
24th December 1998,
Possible diverticulum of the bladder due to leakage.
Possible M.S
5th April 2000
CSF oligoclonal bands have been reported present in the past consistent with demylination pattern.
11-2-00
Gallium scan shows significant area of tracer opaque over left lower hemi-thorax
Presumably lying within the breast in vicinity of nipple.could be underlying breast abscess or carcinoma.
Immunoglobulin G&A minimally reduced.
Acute phase screen haptoglobin,2.03g/l otherwise within normal limits.
Complement cl inhibitor 0.37g/l otherwise within normal limits.
30th Jan 2001
Discharged active problem nuero inflammatory disorder
21st Feb. 2001
Possible nuero-sarcodosis
Bilateral optic neuropathy,
Raised intercranial pressure,
Old anterior uveitis
Possible erythema nodosum
28th June 2001.
Visual fields are constricted bilaterally and visual acuity has deteriorated to N6.
IN MAY 1998 OLIGLONAL BANDS WERE SEEN THEN AGAIN IN NOVEMBER 2000.PRESSURE WAS RAISED ONCE AGAIN TO 34CMSOF C.S.F.
PRESSURE MONITORING SHOWED MILD DEGREE.
24th Jan 2005
symptoms suggest dry eye syndrome, which may reflect evolving lupus.
22ND AUG 2005.
Visual evoked potentials reported to show significant delay in P100 bilaterally.
24th March 2005.
Positive smooth muscle antibodies. Showing vascular pattern associated with wide range of autoimmune diseases.
Possible autoantibodies show changes consistent with a vasculitic disorder.
I was getting treatment for m.s but now doctors are saying possiable lupus.
thankyou for any help.
susan x
rofessor Mitchell?s findings,
22nd may 1998,
Thought to have had vaso-vagal when taken into Furness general in 1998.
Also optic discs swollen more on left than right.
11th June 1998
Oedema of both discs, left side is showing significant field defect and some reduction in colour vision, and VEP showed delayed conduction on both visual pathways.
B scan suggested optic nerve cyst on left side, and possible nerve head drusen on right
22nd June 1998
Thinking the eye problem could be optic nerve head drusen, and pupilay changes may be related to inflammatory uveitis
29th June 1998,
After 15 days in hospital, possible pseudo papilloedema, due to optic head drusen.
Significant field defects on left side, bilateral full discs with blurred margins.
No reaction to light as well as accommodation.
30th July 1998
Visual evoked potentials mildly delayed,
Oligoclonal bands found in CSF and these are absent from the serum.
Raising the possibility of M.S.
9th July 1998
Optic nerve drusen could be anterior segment inflammation.
10th August 1998,
Some indication of demylination. Could be high pressure around optic nerve.
Iris atrophy showing itself as transillumination. Ocular albinism but think its related to chronic inflammation.
Fluoroscein showed leakage from the discs.
11th September 1998.
Pupil abnormality is related to low-grade chronic inflammation.
Possible granulomatous disorder.
24th December 1998,
Possible diverticulum of the bladder due to leakage.
Possible M.S
5th April 2000
CSF oligoclonal bands have been reported present in the past consistent with demylination pattern.
11-2-00
Gallium scan shows significant area of tracer opaque over left lower hemi-thorax
Presumably lying within the breast in vicinity of nipple.could be underlying breast abscess or carcinoma.
Immunoglobulin G&A minimally reduced.
Acute phase screen haptoglobin,2.03g/l otherwise within normal limits.
Complement cl inhibitor 0.37g/l otherwise within normal limits.
30th Jan 2001
Discharged active problem nuero inflammatory disorder
21st Feb. 2001
Possible nuero-sarcodosis
Bilateral optic neuropathy,
Raised intercranial pressure,
Old anterior uveitis
Possible erythema nodosum
28th June 2001.
Visual fields are constricted bilaterally and visual acuity has deteriorated to N6.
IN MAY 1998 OLIGLONAL BANDS WERE SEEN THEN AGAIN IN NOVEMBER 2000.PRESSURE WAS RAISED ONCE AGAIN TO 34CMSOF C.S.F.
PRESSURE MONITORING SHOWED MILD DEGREE.
24th Jan 2005
symptoms suggest dry eye syndrome, which may reflect evolving lupus.
22ND AUG 2005.
Visual evoked potentials reported to show significant delay in P100 bilaterally.
24th March 2005.
Positive smooth muscle antibodies. Showing vascular pattern associated with wide range of autoimmune diseases.
Possible autoantibodies show changes consistent with a vasculitic disorder.
I was getting treatment for m.s but now doctors are saying possiable lupus.
thankyou for any help.
susan x